Research article
PAI-1 and functional blockade of SNAI1 in breast cancer cell migration
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* Corresponding author: Georgia Barlovatz-Meimon georgia.barlovatz@ibisc.fr
- † Equal contributors
1 DYNAMIC Team, IBISC FRE 3190 CNRS – Université d'Evry Val d'Essonne, Genopole, Evry 91000, France
2 Department of Medical Oncology, Hopital Européen Georges Pompidou, Paris cedex 15 75908, France
3 Departamento de Bioquímica, Instituto de Investigaciones Biomedicas 'Alberto Sols', (CSIC-UAM), Madrid 28029, Spain
4 Molecular Pathology Program, Centro Nacional de Investigaciones Oncologicas (CNIO), Madrid 28029, Spain
5 Department of Internal Medicine, University of Michigan School of Medicine, Ann Arbor MI 48109-0644, USA
6 University Paris 12 – Val de Marne, Créteil cedex 94010, France
Breast Cancer Research 2008, 10:R100 doi:10.1186/bcr2203
Published: 3 December 2008Additional files
Additional data file 1:
A table that lists the human genes up-regulated or down-regulated in a clone expressing a dominant-negative form of SNAI1 (SNAI1-DN) compared with MDA-mock control cells. Negative sign (-) means down-regulation and positive sign (+) means up-regulation. Genes are organised by molecular function. A total of 99 genes were found to be differentially expressed, by at least a two-fold factor, in response to SNAI1 functional blockade.
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