An integration of complementary strategies for gene-expression analysis to reveal novel therapeutic opportunities for breast cancer
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* Corresponding author: Andrea H Bild andreab@genetics.utah.edu
- Equal contributors
1 Department of Pharmacology and Toxicology, University of Utah, 112 Skaggs Hall, Salt Lake City, UT 84112, USA
2 Duke Institute for Genome Sciences & Policy, Duke University Medical Center, 2121 CIEMAS, Durham, NC 27701, USA
3 Lineberger Comprehensive Cancer Center, University of North Carolina, 102 Mason Farm Road, Chapel Hill, NC 27599, USA
4 Department of Genetics, University of North Carolina, 120 Mason Farm Road, Chapel Hill, NC 27599, USA
5 The Pulmonary Center, Boston University School of Medicine, 715 Albany St, Boston, MA 02118, USA
6 Department of Pathology & Laboratory Medicine, University of North Carolina, Chapel Hill, NC 27599, USA
7 Division of Hematology/Oncology, Department of Medicine, University of North Carolina, Chapel Hill, NC 27599, USA
8 Carolina Center for Genome Sciences, 5016 Genetic Medicine Building, University of North Carolina, Chapel Hill, NC 27599, USA
Breast Cancer Research 2009, 11:R55 doi:10.1186/bcr2344
Published: 28 July 2009Additional files
Additional file 1:
A box-and-whisker plot showing pathway activation as a function of subtype displayed for an additional validation dataset.
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