Figure 9.

Possible sites of Se-methylselenocysteine (MSC) interaction with components of the Ras–phosphatidylinositol 3-kinase–Akt (Ras–PI3-K–Akt) and Raf–MAP kinase/ERK kinase–ERK (Raf–MEK–ERK) pathways in TM6 mouse mammary tumor cells. MAPK, mitogen-activated protein kinase.