Table 2

Published studies of ovarian suppression with gonadotropin-releasing hormone agonists

Reference
Year
Patients (n)
Chemotherapy regimen
Pregnancies (%)
Births (%)
Menses 1 year after therapy (%)
Menses at the end of follow-up (%)
Study type
Outcome

Fox and colleagues [27]
2003
24
AC, AC-T, FAC, AT-CMF
21
8
96
75
Prospective, single-arm
Ovarian function preservation
Del Mastro and colleagues [28]
2006
29
100% FEC
-
-
94
92
Prospective, single-arm
Ovarian function preservation
Recchia and colleagues [29]
2002
100
26% CMF, 11% FEC, 54% CMF + epirubicin, 9% HCST
3
2
100

Retrospective, single-arm
Ovarian function preservation
Urruticoechea and colleagues [30]
2007
50
78% FEC, 14% AC, 8% AC-T/D
16
16
86
90
Prospective, single-arm
Ovarian function preservation

Total

203








AC, adriamycin (doxorubicin), and cyclophosphamide; AC-T, adriamycin (doxorubicin), cyclophosphamide and taxol (paclitaxel); AC-T/D, adriamycin (doxorubicin), cyclophosphamide and taxol (paclitaxel)/docetaxel; AT-CMF, adriamycin (doxorubicin), taxol (paclitaxel), cyclophosphamide, methotrexate, and 5-fluorouracil; CMF, cyclophosphamide, methotrexate, and 5-fluorouracil; FAC, 5-fluorouracil, adriamycin (doxorubicin), and cyclophosphamide; FEC, 5-fluorouracil, epirubicin, and cyclophosphamide; HCST, high dose chemotherapy and autologous peripheral blood progenitor cell transplantation.

Maltaris et al. Breast Cancer Research 2008 10:206   doi:10.1186/bcr1991